Correction: Interactome analysis of the lymphocytic choriomeningitis virus nucleoprotein in infected cells reveals ATPase Na+/K+ transporting subunit Alpha 1 and prohibitin as host-cell factors involved in the life cycle of mammarenaviruses
																	
									Autoři:
											Masaharu Iwasaki; 											Petra Minder; 											Yíngyún Caì; 											Jens H. Kuhn; 											John R. Yates, III; 											Bruce E. Torbett; 											Juan C. de la Torre										
				
				
									Vyšlo v časopise:
					Correction: Interactome analysis of the lymphocytic choriomeningitis virus nucleoprotein in infected cells reveals ATPase Na+/K+ transporting subunit Alpha 1 and prohibitin as host-cell factors involved in the life cycle of mammarenaviruses. PLoS Pathog 16(1): e1008289. doi:10.1371/journal.ppat.1008289
					
				
									Kategorie:
					Correction
					
				
									prolekare.web.journal.doi_sk:
					
						https://doi.org/10.1371/journal.ppat.1008289
					
							
Zdroje
1. Iwasaki M, Minder P, Caì Y, Kuhn JH, Yates JR III, Torbett BE, et al. (2018) Interactome analysis of the lymphocytic choriomeningitis virus nucleoprotein in infected cells reveals ATPase Na+/K+ transporting subunit Alpha 1 and prohibitin as host-cell factors involved in the life cycle of mammarenaviruses. PLoS Pathog 14(2): e1006892. https://doi.org/10.1371/journal.ppat.1006892 29462184
Štítky
Hygiena a epidemiológia Infekčné lekárstvo LaboratóriumČlánok vyšiel v časopise
PLOS Pathogens
2020 Číslo 1
- Parazitičtí červi v terapii Crohnovy choroby a dalších zánětlivých autoimunitních onemocnění
- Očkování proti virové hemoragické horečce Ebola experimentální vakcínou rVSVDG-ZEBOV-GP
- Koronavirus hýbe světem: Víte jak se chránit a jak postupovat v případě podezření?
Najčítanejšie v tomto čísle
- Correction: Interactome analysis of the lymphocytic choriomeningitis virus nucleoprotein in infected cells reveals ATPase Na+/K+ transporting subunit Alpha 1 and prohibitin as host-cell factors involved in the life cycle of mammarenaviruses
- A shear stress micromodel of urinary tract infection by the Escherichia coli producing Dr adhesin
- USP12 translocation maintains interferon antiviral efficacy by inhibiting CBP acetyltransferase activity
- Norovirus infection results in eIF2α independent host translation shut-off and remodels the G3BP1 interactome evading stress granule formation