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Correction: Interactome analysis of the lymphocytic choriomeningitis virus nucleoprotein in infected cells reveals ATPase Na+/K+ transporting subunit Alpha 1 and prohibitin as host-cell factors involved in the life cycle of mammarenaviruses
Authors: Masaharu Iwasaki; Petra Minder; Yíngyún Caì; Jens H. Kuhn; John R. Yates, III; Bruce E. Torbett; Juan C. de la Torre
Published in the journal: Correction: Interactome analysis of the lymphocytic choriomeningitis virus nucleoprotein in infected cells reveals ATPase Na+/K+ transporting subunit Alpha 1 and prohibitin as host-cell factors involved in the life cycle of mammarenaviruses. PLoS Pathog 16(1): e1008289. doi:10.1371/journal.ppat.1008289
Category: Correction
doi: https://doi.org/10.1371/journal.ppat.1008289The following information is missing from the Funding statement: This research was supported by the US National Institute of General Medical Sciences (NIGMS) (https://www.nigms.nih.gov/) grant U54 GM103368 to BET, and the US National Institute of Allergy and Infectious Diseases (NIAID) (https://www.niaid.nih.gov/) grant U54 AI150472 to BET.
Zdroje
1. Iwasaki M, Minder P, Caì Y, Kuhn JH, Yates JR III, Torbett BE, et al. (2018) Interactome analysis of the lymphocytic choriomeningitis virus nucleoprotein in infected cells reveals ATPase Na+/K+ transporting subunit Alpha 1 and prohibitin as host-cell factors involved in the life cycle of mammarenaviruses. PLoS Pathog 14(2): e1006892. https://doi.org/10.1371/journal.ppat.1006892 29462184
Štítky
Hygiena a epidemiológia Infekčné lekárstvo Laboratórium
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Najčítanejšie v tomto čísle- Correction: Interactome analysis of the lymphocytic choriomeningitis virus nucleoprotein in infected cells reveals ATPase Na+/K+ transporting subunit Alpha 1 and prohibitin as host-cell factors involved in the life cycle of mammarenaviruses
- A shear stress micromodel of urinary tract infection by the Escherichia coli producing Dr adhesin
- USP12 translocation maintains interferon antiviral efficacy by inhibiting CBP acetyltransferase activity
- Norovirus infection results in eIF2α independent host translation shut-off and remodels the G3BP1 interactome evading stress granule formation
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