Correction: The bZIP Transcription Factor MoAP1 Mediates the Oxidative Stress Response and is Critical for Pathogenicity of the Rice Blast Fungus Magnaporthe oryzae
Authors:
Min Guo; Yue Chen; Yan Du; Yanhan Dong; Wang Guo; Su Zhai; Haifeng Zhang; Suomeng Dong; Zhengguang Zhang; Yuanchao Wang; Ping Wang; Xiaobo Zheng
Published in the journal:
Correction: The bZIP Transcription Factor MoAP1 Mediates the Oxidative Stress Response and is Critical for Pathogenicity of the Rice Blast Fungus Magnaporthe oryzae. PLoS Pathog 15(11): e32767. doi:10.1371/journal.ppat.1008196
Category:
Correction
doi:
https://doi.org/10.1371/journal.ppat.1008196
The authors would like to correct images in S12 Fig. In S12A and S12B Fig, duplicate images were mistakenly illustrated during the preparation of figures for publication. The authors have now repeated the experiments and provided the new images. The authors confirm that these changes do not alter any findings.
Supporting information
S12 Fig [a]
Pathogenicity test of the down-regulated gene deletion mutants.
Zdroje
1. Guo M, Chen Y, Du Y, Dong Y, Guo W, Zhai S, et al. (2011) The bZIP Transcription Factor MoAP1 Mediates the Oxidative Stress Response and Is Critical for Pathogenicity of the Rice Blast Fungus Magnaporthe oryzae. PLoS Pathog 7(2): e1001302. https://doi.org/10.1371/journal.ppat.1001302 21383978
Štítky
Hygiena a epidemiológia Infekčné lekárstvo LaboratóriumČlánok vyšiel v časopise
PLOS Pathogens
2019 Číslo 11
- Parazitičtí červi v terapii Crohnovy choroby a dalších zánětlivých autoimunitních onemocnění
- Očkování proti virové hemoragické horečce Ebola experimentální vakcínou rVSVDG-ZEBOV-GP
- Koronavirus hýbe světem: Víte jak se chránit a jak postupovat v případě podezření?
Najčítanejšie v tomto čísle
- Candida albicans triggers NADPH oxidase-independent neutrophil extracellular traps through dectin-2
- Mycobacterium abscessus virulence traits unraveled by transcriptomic profiling in amoeba and macrophages
- Trickle infection and immunity to Trichuris muris
- Porphyromonas gingivalis induces penetration of lipopolysaccharide and peptidoglycan through the gingival epithelium via degradation of junctional adhesion molecule 1