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PLOS Pathogens - Číslo 4/2011

Pearls

32767

Innate Immune Sensing of DNA

Review

32767

Genetic Assignment Methods for Gaining Insight into the Management of Infectious Disease by Understanding Pathogen, Vector, and Host Movement

Research Article

32767

Low Diversity Variety Multilocus Sequence Types from Thailand Are Consistent with an Ancestral African Origin

Engineered Resistance to Development in Transgenic

-Mediated Detoxification of Reactive Oxygen Species Is Required for Full Virulence in the Rice Blast Fungus

SLO-1-Channels of Parasitic Nematodes Reconstitute Locomotor Behaviour and Emodepside Sensitivity in Loss of Function Mutants

Structure-Function Analysis of the LRIM1/APL1C Complex and its Interaction with Complement C3-Like Protein TEP1

A Effector with Enhanced Inhibitory Activity on the NF-κB Pathway Activates the NLRP3/ASC/Caspase-1 Inflammasome in Macrophages

The MARCH Family E3 Ubiquitin Ligase K5 Alters Monocyte Metabolism and Proliferation through Receptor Tyrosine Kinase Modulation

is an Unstable Pathogen Showing Evidence of Significant Genomic Flux

The Cell Wall Protein CwpV is Antigenically Variable between Strains, but Exhibits Conserved Aggregation-Promoting Function

Engineering HIV-Resistant Human CD4+ T Cells with CXCR4-Specific Zinc-Finger Nucleases

SUMO-Interacting Motifs of Human TRIM5α are Important for Antiviral Activity

Completion of Hepatitis C Virus Replication Cycle in Heterokaryons Excludes Dominant Restrictions in Human Non-liver and Mouse Liver Cell Lines

: Reservoir Hosts and Tracking the Emergence in Humans and Macaques

On Being the Right Size: The Impact of Population Size and Stochastic Effects on the Evolution of Drug Resistance in Hospitals and the Community

Bacterial and Host Determinants of MAL Activation upon EPEC Infection: The Roles of Tir, ABRA, and FLRT3

Respiratory Syncytial Virus Interferon Antagonist NS1 Protein Suppresses and Skews the Human T Lymphocyte Response

NF-κB Hyper-Activation by HTLV-1 Tax Induces Cellular Senescence, but Can Be Alleviated by the Viral Anti-Sense Protein HBZ

Human Cytomegalovirus IE1 Protein Elicits a Type II Interferon-Like Host Cell Response That Depends on Activated STAT1 but Not Interferon-γ

A New Model to Produce Infectious Hepatitis C Virus without the Replication Requirement

PLOS Pathogens

2011 Číslo 4

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